منابع مشابه
Drug-induced immune complex disease
19. Lenk, R. P., Maize], J. V. & Crouch, R. J. ( 1 982) Eur. J. Biochem. 121,475-482 Habets, W. J., Moet, M., van de Pas, J. & Van Venrooij, W. J. (1985) in Protides of the Biological Fluids (Peeters, H., ed.), vol. 33, pp. 199-204 Baboonian, C., Venables, P. J. W., Booth, J., Williams, D. G., Roffe, L. M. & Maini, R N. (1989) Clin. Exp. Immunol. 78,454-459 Mathews, M. B. & Bernstein, R. M. (19...
متن کاملAcute Immune Complex Disease in Rabbits
By depletion of C3 from rabbits undergoing acute experimental immune complex disease with an anticomplementary factor in cobra venom, it has been possible to demonstrate that deposition of the complexes in arteries and glomeruli does not require the complement components reacting after C2. Immunological reactions, in which platelets release their vasoactive amines, have been examined in rabbits...
متن کاملExperimental Immune Complex Disease of the Lung
Membranous and/or proliferative pneumonitis, similar in certain features to human interstitial pneumonitis, developed in rabbits making hyperactive antibody response to daily injections of bovine serum albumin (BSA) administered in multiple large doses sufficient to maintain the state of relative antigen-antibody equivalence. The pulmonary lesions were associated with deposition in alveolar cap...
متن کاملNeonatal Fc receptor promotes immune complex-mediated glomerular disease.
The neonatal Fc receptor (FcRn) is a major regulator of IgG and albumin homeostasis systemically and in the kidneys. We investigated the role of FcRn in the development of immune complex-mediated glomerular disease in mice. C57Bl/6 mice immunized with the noncollagenous domain of the α3 chain of type IV collagen (α3NC1) developed albuminuria associated with granular capillary loop deposition of...
متن کاملDeficiency in immune complex uptake by chronic granulomatous disease neutrophils.
Receptor-mediated binding and endocytosis by normal and chronic granulomatous disease (CGD) polymorphonuclear (PMN) leucocytes have been investigated. Our results suggest that activation of the respiratory burst is a requisite for normal immune complex internalization. Cell-associated immune complexes were detected by fluorescence microscopy, electron microscopy and flow cytometric techniques. ...
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ژورنال
عنوان ژورنال: Annals of the Rheumatic Diseases
سال: 1973
ISSN: 0003-4967
DOI: 10.1136/ard.32.3.265